as we manage to 'see' in ways not necessarily determined by their design.
of course we do. We compensate for hardware problems with software. That's why you're not even aware of your blind spot.
But. Your blind spot is there. There's a huge whole in your perception. Just because you can't see it doesn't make it any better.
And what else does the software make up for? The fact that you only have clear vision in the fovea of your retina and that the rest of your vision only seems to be clear because of the software?
How about the lack of color vision outside of the fovea? Amazing that. That you only really see colors in a small portion of your vision but think that you see color everywhere.
There's a million and one little things that are weird about the human eye. Its defects made up for elsewhere.
How about we take a step back for a second. Ok?
Let's look at your question.
You want an explanation for the human eye.
Do you even realize how nonsensical that question is? What do you want explained about it? What 'evidence' are you looking for?
Do you want a 1000 page essay as an abstract for the outline of such an endeavor?
How about we start with one simple fact of the evolution of the eye? Ok?
Humans have three cones. Yes? Reptiles have four. Because of this, they can see in the ultraviolet. Did you know that? They can.
Not only this, but we only share two of our cones with them. And some of the earlier mammals only have two cones.
Here's the story. Early mammals were nocturnal. Color vision at night is not really very useful. So, a mutation or three caused the jettisoning of two of the cones.
Ok?
Well. A few million years later, mammals were diurnal as well as nocturnal. This means that those extra cones would come in handy. But, they were gone.
Well. A beneficial duplication duplicated one of the cones. Not a precise duplication, but one which reacts to a pretty similar bandwidth of frequencies. This isn't the best solution. Far from. But it works better than two.
So. Here we are.
You might also find it interesting to note that the duplicated gene was on the X chromosome. This is why its men who are more prone to color blindness. Because we only have one copy of the X. So if we get an X with a crappy cone gene that places the frequency spread too close to each other? We can't seperate colors very well.
Yay for the eye! Woohoo!