That doesn't contradict what I said about the evolutionary advantage that Marsupials clearly have over Placentals in Australia.We don't know everything.. so keep your shit to yourself that there is no benefit. Maybe there is. Do you know that there isn't? Perhaps we should end science now :shrug:
No, it is immunosuppression. Plain and simple. Trying to dress it up as anything else is pure obfuscation.So although it was to protect the embryo in case of infection the EMBRYO initiates the mother to respond- saving the mother most likely and putting the embryo at risk! Isn't that just coordination- that is what the suppression would be called in the scheme of things.
It contains foreign proteins, foreign antigens, so from the bodies perspective, it is a foreign object - which is the only thing I have ever actually stated in this thread, although you have managed to infer some truely bizzare things from that statement. All of this bleeding heart nonsense of yours is just trying to dress it up as something that it isn't.Cells don't 'know shit, they simply react to stimulus' so it makes sense to suppress the maternal immune system- but that is part of the internalized process. In the overall scheme of things it was never 'foreign' (as in a parasite) it was 'foreign' only in the technical terms that it not the same cell as the mother. But its 'development' being coordinated by other can't be labeled 'foreign'-
If embryo was just a parasite, initiating the mother's immune system in a virus would be self-destruction. Not a parasitic activity
More obfuscatory drivel. From your own source:
All that exquisitely synchronized activity can be derailed by viral or bacterial infection. Clinical studies have shown that infections cause as much as 40 percent of incidence of preterm labor. Furthermore, of the most severe cases of preterm delivery (pregnancies that end after less than thirty weeks of gestation), 80 percent show evidence of infection...
...To our surprise, my research group discovered that the signals triggering miscarriage may be initiated by the same guest Conductor, the trophoblast. If a virus, say, is infecting the uterus, the trophoblast recognizes the virus through its TLRs, just as in normal implantation the trophoblast recognizes dead cells from the mother's uterus. With the virus, however, the trophoblast's response is different. Its cells signal the mother's immune-system cells to mount an aggressive immune response. Instead of suppressing her cytolytic T cells and natural killer cells, she activates them. Not only do they attack the infection, but they attack the trophoblast as well. The battered embryonic tissue is then expelled; the mother miscarries.
An infection induced miscarriage occurs when the trophoblast stops suppressing the maternal immune response, and antagonizes it into rejecting it as a foreign body....To our surprise, my research group discovered that the signals triggering miscarriage may be initiated by the same guest Conductor, the trophoblast. If a virus, say, is infecting the uterus, the trophoblast recognizes the virus through its TLRs, just as in normal implantation the trophoblast recognizes dead cells from the mother's uterus. With the virus, however, the trophoblast's response is different. Its cells signal the mother's immune-system cells to mount an aggressive immune response. Instead of suppressing her cytolytic T cells and natural killer cells, she activates them. Not only do they attack the infection, but they attack the trophoblast as well. The battered embryonic tissue is then expelled; the mother miscarries.